Elevated levels of sIL-2R, neopterin, and beta-2 microglobulin in idiopathic dilated cardiomyopathy appear to reflect heart failure severity and advanced disease rather than a specific autoimmune pathogenesis.
No takes yet. Share an insight, caveat, or question.
May reflect HF severity over autoimmune etiology in idiopathic DCM; hypothesis-generating and should not change practice.
Caforio et al. (2001) studied this question.
Synapse has enriched 2 closely related papers on similar clinical questions. Consider them for comparative context: