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April 1, 2003Archives of Neurology

M-Channels

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Authors

ECEdward C. CooperBaylor College of Medicine
Lily Yeh Jan
Lily Yeh JanElectrophysiology

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Implication

May guide M-channel drug development for epilepsy; leaves open clinical translation in stroke and Alzheimer disease.

Key Points

Key points are not available for this paper at this time.

Cite This Study

Cooper et al. (2003) studied this question.

synapsesocial.com/papers/6a7c25a2d501feb4353bd7fahttps://doi.org/10.1001/archneur.60.4.496
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Colocalization and coassembly of two human brain M-type potassium channel subunits that are mutated in epilepsy2000 · 219 citations
  2. 2A Potassium Channel Mutation in Neonatal Human Epilepsy1998 · 1,089 citations
  3. 3Myokymia and neonatal epilepsy caused by a mutation in the voltage sensor of the KCNQ2 K <sup>+</sup> channel2001 · 255 citations
  4. 4M Channel KCNQ2 Subunits Are Localized to Key Sites for Control of Neuronal Network Oscillations and Synchronization in Mouse Brain2001 · 306 citations
  5. 5Ion Channels in Drosophila1988 · 41 citations