This study identifies a novel PKCdelta-Thr(505) autophosphorylation mechanism triggered by PKCepsilon overexpression and involving Src-dependent phosphorylation in cardiomyocytes.
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Hypothesis-generating for PKC isoform crosstalk in cardiomyocytes; extends mechanistic models but leaves open human relevance.
Rybin et al. (2007) studied this question.
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