Population
Juxtamedullary tissue from captopril-treated rats perfused with blood at a constant pressure of 110 mmHg
Design
Preclinical
Authors
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Physiological ANG II vasoconstricts juxtamedullary vessels in rodents; leaves open translation to human renal hemodynamics and hypertension.
Physiological concentrations of angiotensin II elicit receptor-dependent and reversible vasoconstriction of the juxtamedullary nephron microvasculature at both pre- and postglomerular sites.
Carmines et al. (1986) studied this question.
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