High plasma concentrations of chylomicron-like triglyceride emulsions compete efficiently with liver-derived VLDL for lipolysis, leading to the accumulation of endogenous VLDL in the postprandial state.
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Chylomicron-VLDL lipolytic competition may drive postprandial endogenous VLDL accumulation; leaves open human relevance and therapeutic implications.
Karpe et al. (1995) studied this question.
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