Why the study?
While anticoagulation with VKAs and DOACs prevents stroke in AF, their role in reducing dementia risk remains unclear.
Does anticoagulation therapy, particularly DOACs, reduce the incidence of dementia in patients with atrial fibrillation compared to VKAs or no anticoagulation?
Does anticoagulation therapy, particularly DOACs, reduce the incidence of dementia in patients with atrial fibrillation compared to VKAs or no anticoagulation?
Anticoagulation therapy, particularly DOACs, may reduce the risk of incident dementia in patients with atrial fibrillation compared to VKAs, though current evidence is of moderate to low certainty.
Should not yet change AF practice; extends observational support for OAC in dementia prevention.
Atrial fibrillation (AF) increases the risk of stroke and cognitive decline. While anticoagulation with vitamin K antagonists (VKAs) and direct oral anticoagulants (DOACs) prevents stroke, their role in reducing dementia risk in patients with AF remains unclear. To evaluate the effect of anticoagulation therapy on dementia incidence in patients with AF, comparing DOACs versus VKAs. We systematically reviewed PubMed, Scopus, Web of Science, Embase, and Cochrane. Systematic reviews and meta-analyses evaluating the effects of anticoagulation therapies on dementia were included. A total of 11 systematic reviews and meta-analyses were included in this umbrella review. Findings from 6 systematic reviews showed that OAC use was associated with a reduced risk of incident dementia in patients with AF, with effect estimates (RR/HR) ranging from 0.46 [0.28-0.78] to 0.79 [0.67-0.93]. For DOACs versus VKAs, most studies found a lower risk of dementia with DOACs, with effect sizes ranging from HR: 0.51 [0.37-0.71] to RR: 0.88 [0.82-0.94]. However, two studies found no significant difference between DOACs and warfarin in dementia risk (OR: 0.65 [0.34-1.25] and RR: 0.91 [0.75-1.12], respectively). Anticoagulation therapy, particularly with DOACs, may help reduce the risk of dementia in AF patients. The evidence remains of moderate to low certainty, and further high-quality, long-term randomized controlled trials are needed to confirm these findings and explore the neuroprotective mechanisms of OACs.
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Afzalnia et al. (2026) studied this question.
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