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February 1, 2022BiomedicinesOpen Access

Mitochondrial Metabolism, Redox, and Calcium Homeostasis in Pulmonary Arterial Hypertension

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Why the study?

Metabolic reprogramming plays a critical role in pulmonary arterial hypertension pathogenesis, and understanding mitochondrial metabolic mechanisms could provide viable therapeutic approaches.

Design

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Authors

SLShuxin LiangMYManivannan YegambaramTWTing Wang

Discussion

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Overview

Mitochondrial pathway modulation should not yet change PAH practice; leaves open whether it extends current vasodilator strategies.

Structured PICO

P
Population
Pulmonary arterial hypertension (PAH)

Understanding mitochondrial metabolic mechanisms in PAH could provide viable therapeutic approaches for patients.

Cite This Study

Liang et al. (2022) studied this question.

synapsesocial.com/papers/6a7fac93c32ce294b5d76e56https://doi.org/10.3390/biomedicines10020341
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Mitochondrial dysfunction in pulmonary arterial hypertension2022 · 33 citations
  2. 2Metabolism and Bioenergetics in the Right Ventricle and Pulmonary Vasculature in Pulmonary Hypertension2013 · 179 citations
  3. 3Metabolic Alterations Associated With Right Ventricular Dysfunction in Pulmonary Arterial Hypertension: The Modulatory Effects and Improvement Mechanisms of Exercise2025 · 5 citations
  4. 4The Basic Science of Metabolism in Pulmonary Arterial Hypertension2018 · 10 citations
  5. 5Metabolic reprogramming: A novel metabolic model for pulmonary hypertension2022 · 16 citations