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August 28, 2008Clinical and Experimental Pharmacology and Physiology

Potential Role of Nitric Oxide in Contraction‐stimulated Glucose Uptake and Mitochondrial Biogenesis in Skeletal Muscle

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GMGlenn K. McConellUniversity of Copenhagen GWGlenn D. WadleyDeakin University

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Implication

Supports NO regulation of exercise glucose uptake; leaves open NO-targeted glycemic therapies in diabetes.

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Key points are not available for this paper at this time.

Cite This Study

McConell et al. (2008) studied this question.

synapsesocial.com/papers/6a800ad7ccc36c0bd6ee9dde https://doi.org/10.1111/j.1440-1681.2008.05038.x

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Nitric oxide synthase inhibition reduces leg glucose uptake but not blood flow during dynamic exercise in humans.1999 · 170 citations
2Role of the nitric oxide pathway in AMPK-mediated glucose uptake and GLUT4 translocation in heart muscle2004 · 198 citations
3nNOS and eNOS modulate cGMP formation and vascular response in contracting fast-twitch skeletal muscle2000 · 157 citations
4Modular Organization of T4 DNA Polymerase1995 · 95 citations
5Nitric oxide release is present from incubated skeletal muscle preparations1994 · 405 citations