Population
Arterial smooth muscle
Design
Preclinical
Authors
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May alter vascular excitability in animal models; leaves open relevance to human hypertension or therapy.
This study identifies a novel signaling cascade where Angiotensin II regulates arterial smooth muscle excitability by decreasing Kv2.1 expression via a calcineurin and NFATc3-dependent pathway.
Amberg et al. (2004) studied this question.
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