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March 19, 2004The FASEB Journal

Atrial natriuretic peptide contributes to the physiological control of lipid mobilization in humans

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Why the study?

Does acute oral tertatolol treatment affect exercise-induced lipid mobilization and ANP release in healthy young men?

Population

Healthy young men

Comparison

Acute oral tertatolol treatment prior to two… vs Placebo prior to exercise bouts

Design

Other

Authors

CMCédric MoroInsermFCF. CrampesInsermCSCoralie SengenèsNEOMA Business School

Discussion

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Implication

Beta-blockade preserves exercise lipolysis while amplifying ANP; confirms ANP as a potentiated, adrenergic-independent mediator of fat mobilization.

Structured PICO

Does acute oral tertatolol treatment affect exercise-induced lipid mobilization and ANP release in healthy young men?

P
Population
Healthy young men
I
Intervention
Acute oral tertatolol (nonselective beta-antagonist) treatment prior to two successive exercise bouts at 35% and 60% peak oxygen consumption (VO2max)
C
Comparator
Placebo prior to exercise bouts
O
Outcome
Extracellular glycerol concentration (EGC) in subcutaneous adipose tissue measured by in situ microdialysissurrogate

Exercise-induced lipid mobilization in humans is partially mediated by atrial natriuretic peptide, a pathway that is potentiated by oral beta-adrenergic blockade.

Cite This Study

Moro et al. (2004) studied this question.

synapsesocial.com/papers/6a821658ceb2bc8b5096fc40https://doi.org/10.1096/fj.03-1086fje
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Adrenergic regulation of lipolysis in situ at rest and during exercise.1990 · 373 citations
  2. 2Parathyroid hormone-induced lipolysis in human adipose tissue1987 · 37 citations
  3. 3Gender Differences in Adremergic Regulation of Lipid Mobilization During Exercise1996 · 101 citations
  4. 4Exercise metabolism in healthy volunteers taking celiprolol, atenolol, and placebo.1997 · 16 citations