The study suggests that SLE-prone mice produce antibodies against their own xenotropic viral gp70 due to immunologic dysfunction rather than the expression of an unusual gp70 type.
No takes yet. Share an insight, caveat, or question.
Immune dysregulation likely drives anti-gp70 responses in SLE mice; leaves open relevance to human lupus pathogenesis.
Izui et al. (1981) studied this question.
Synapse has enriched 3 closely related papers on similar clinical questions. Consider them for comparative context: