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April 1, 2001The Neuroscientist

Maxi-K Potassium Channels: Form, Function, and Modulation of a Class of Endogenous Regulators of Intracellular Calcium

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VGValentin K. GribkoffUniversity of California, Riverside JSJohn E. StarrettPennsylvania State University SDSteven I. DworetzkyBiohaven Pharmaceuticals (United States)

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Implication

May support maxi-K modulator development for stroke and cognition; leaves open validation in prospective clinical trials.

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Key points are not available for this paper at this time.

Cite This Study

Gribkoff et al. (2001) studied this question.

synapsesocial.com/papers/6a82c5b6abc87fc4bf1e1da5 https://doi.org/10.1177/107385840100700211

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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

1Opening of large-conductance calcium-activated potassium channels by the substituted benzimidazolone NS0041994 · 68 citations
2BK Channel Activation by Brief Depolarizations Requires Ca2+ Influx Through L- and Q-Type Ca2+ Channels in Rat Chromaffin Cells1999 · 104 citations
3Activation of BKCa channels in acutely dissociated neurones from the rat ventromedial hypothalamus by NS 16191994 · 33 citations
4Cloning and characterization of human and mouse homologs of the Drosophila calcium-activated potassium channel gene, slowpoke1994 · 179 citations