Population
Smooth muscle cells dispersed by enzymatic treatment from rat portal veins
Design
Preclinical
Authors
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Hypothesis-generating for K-channel modulation in vascular smooth muscle; leaves open human translation and clinical relevance.
Levcromakalim may induce a voltage-independent K-current in rat portal vein smooth muscle cells by dephosphorylating the delayed rectifier channel, converting it into a KATP form.
Edwards et al. (1993) studied this question.
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