Key result
Homozygous factor V Leiden carriers had a significantly increased risk of pregnancy-related venous thrombosis compared to women with normal coagulation (15.8% vs 0.5%; RR 41.3, 95% CI 4.1-419.7).
Why the study?
Does severe inherited thrombophilia (homozygous factor V Leiden or double heterozygous mutations) increase the risk of pregnancy-related venous thrombosis?
Population
236 pregnant women, including 15 homozygous factor V Leiden, 39 double heterozygous, and 182 with normal…
Comparison
Homozygous factor V Leiden or double… vs Normal coagulation
Design
Cohort
Follow-up
Pregnancy and postpartum period
Authors
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May support individualized prophylaxis decisions in pregnancy; extends observational risk data but leaves open need for RCT confirmation.
Cohort (n=236)
Yes
Does severe inherited thrombophilia (homozygous factor V Leiden or double heterozygous mutations) increase the risk of pregnancy-related venous thrombosis?
Relative Risk: 41.3 (95% CI 4.1–419.7)
Absolute Event Rate: 15.8% vs 0.5%
Homozygous factor V Leiden and double heterozygous carriers have a significantly increased risk of venous thrombosis during pregnancy and postpartum, supporting the need for anticoagulant prophylaxis.
Legnani et al. (2001) conducted a cohort in Pregnancy-related venous thrombosis (n=236). Homozygous factor V Leiden vs. Normal coagulation was evaluated on Pregnancy-related venous thrombosis (RR 41.3, 95% CI 4.1-419.7). Homozygous factor V Leiden carriers had a significantly increased risk of pregnancy-related venous thrombosis compared to women with normal coagulation (15.8% vs 0.5%; RR 41.3, 95% CI 4.1-419.7).
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