Why the study?
What are the characteristics and clinical effects of myopotential signals in patients with unipolar DDD pacemakers?
What are the characteristics and clinical effects of myopotential signals in patients with unipolar DDD pacemakers?
Myopotentials in unipolar DDD pacemakers frequently cause ventricular tracking or inhibition, but these effects can be effectively managed through pacemaker reprogramming guided by electrogram telemetry.
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Myopotential sensing may be mitigated by reprogramming in unipolar DDD pacemakers; leaves open need for prospective outcome studies.
Myopotential signals were recorded from atrial and ventricular leads during isometric exercise in 25 patients who had chronically implanted dual chamber pacemakers using the electrogram telemetry capability of the pacemakers. Average electrogram amplitude on the atrial channel was 0.92 mV (range 0.3 to 1.9) and on the ventricular channel was 0.98 mV (range 0.3 to 2.2); the difference was not significant. There was a strong correlation (R = 0.82) between the amplitude of myopotentials on the atrial and ventricular leads for individual patients. Myopotential sensing caused ventricular output inhibition in two patients (8%) and ventricular tracking in sixteen patients (64%). Pacemaker reprogramming abolished ventricular myopotential inhibition in all patients and stopped ventricular myopotential tracking in seven patients. We conclude that myopotentials can be analyzed and their effects ameliorated by a multiprogrammable pacemaker with electrogram telemetry capability.
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Zimmern et al. (1986) studied this question.
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