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Does aerobic exercise training alter sympathetic vasoconstrictor responsiveness and enhance functional sympatholysis through a nitric oxide-dependent mechanism?
Does aerobic exercise training alter sympathetic vasoconstrictor responsiveness and enhance functional sympatholysis through a nitric oxide-dependent mechanism?
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This article reviews the hypothesis that aerobic exercise training modulates sympathetic vasoconstriction and enhances functional sympatholysis via a nitric oxide-dependent mechanism.
Sympathetic vasoconstriction in the skeletal muscle vascular bed is essential for the regulation of vascular resistance and therefore control of blood pressure and muscle blood flow at rest and during exercise. In this article, we address the hypothesis that aerobic exercise training alters sympathetic vasoconstrictor responsiveness and enhances contraction-mediated inhibition of sympathetic vasoconstriction (functional sympatholysis) through a nitric oxide–dependent mechanism.
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Just et al. (2016) studied this question.
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