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December 1, 1997AJP Renal Physiology

G protein mRNA expression in renal microvessels from spontaneously hypertensive and Wistar-Kyoto rats

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Authors

RMRupa MokkapattiUniversity of Pittsburgh SVSubhash J. VyasUniversity of Pittsburgh EJEdwin K. JacksonCross-Cutting Cardiology

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Implication

Altered G-protein balance may underlie renal ANG II hypersensitivity in SHR; leaves open human relevance and therapeutic translation.

Key Points

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Cite This Study

Mokkapatti et al. (1997) studied this question.

synapsesocial.com/papers/6a856bcf425e2f70a4671756 https://doi.org/10.1152/ajprenal.1997.273.6.f877

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Enhanced expression of inhibitory guanine nucleotide regulatory protein in spontaneously hypertensive rats. Relationship to adenylate cyclase inhibition1992 · 132 citations
2G-proteins in experimental hypertension: a study of spontaneously hypertensive rat myocardial and renal cortical plasma membranes1993 · 31 citations
3Defective G protein activation of the cAMP pathway in rat kidney during genetic hypertension.1995 · 73 citations
4Cloning of the alpha-subunit of GS protein from spontaneously hypertensive rats.1994 · 7 citations
5Differential pressor and renal vascular reactivity to angiotensin II in spontaneously hypertensive and Wistar-Kyoto rats.1987 · 27 citations