Population
Spontaneously hypertensive rat (SHR) model of genetic hypertension and Wistar-Kyoto (WKY) rats
Comparison
Cloning, sequencing, and expression analysis of… vs Wistar-Kyoto (WKY) rats
Design
Preclinical
Authors
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Intact alpha S in SHR excludes GS subunit defect as cause of Na+,K(+)-ATPase dysregulation; leaves open other mechanisms for hypertension research.
The abnormal regulation of Na+,K(+)-ATPase activity in spontaneously hypertensive rats is not caused by a defect in the alpha S subunit of the GS protein.
Gurich et al. (1994) studied this question.
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