Population
Human subjects with the genetic disorder abetalipoproteinemia, normal human subjects, and patients with…
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Editorial
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The absence of the 88-kD subunit of microsomal triglyceride transfer protein explains the inability to form and secrete apolipoprotein B-containing lipoproteins in abetalipoproteinemia.
The absence of the 88-kD subunit of microsomal triglyceride transfer protein explains the inability to form and secrete apolipoprotein B-containing lipoproteins in abetalipoproteinemia.
Hamilton et al. (1993) studied this question.
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