Identifies a specific 10-residue motif centered around Thr53 in the N terminus of the Kir2.3 channel responsible for its gating by intracellular protons.
No takes yet. Share an insight, caveat, or question.
Should not yet alter arrhythmia management; extends molecular insights into Kir2.3 gating in animal models.
Qu et al. (1999) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: