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September 1, 1998AJP Renal Physiology

Neuronal nitric oxide synthase is expressed in principal cell of collecting duct

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Authors

XWXianhong WangQingdao University of Science and Technology MLMing LuHubei University of Arts and Science YGYongzong Gao

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Implication

nNOS in CCD may regulate K+ secretion; leaves open its functional role and clinical relevance in potassium disorders.

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Key points are not available for this paper at this time.

Cite This Study

Wang et al. (1998) studied this question.

synapsesocial.com/papers/6a88eb798642fe04be696be3 https://doi.org/10.1152/ajprenal.1998.275.3.f395

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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Topography of nitric oxide synthesis by localizing constitutive NO synthases in mammalian kidney1995 · 363 citations
2Polymerase chain reaction localization of constitutive nitric oxide synthase and soluble guanylate cyclase messenger RNAs in microdissected rat nephron segments.1992 · 246 citations
3Endothelium-derived relaxing factor inhibits transport and increases cGMP content in cultured mouse cortical collecting duct cells.1992 · 125 citations
4Neuronal Nitric-oxide Synthase-μ, an Alternatively Spliced Isoform Expressed in Differentiated Skeletal Muscle1996 · 352 citations
5ANF and bradykinin synergistically inhibit transport in M-1 cortical collecting duct cell line1992 · 19 citations