Why the study?
Does CYP2J2 overexpression or exogenous EETs reduce oxidative stress and apoptosis in lung ischemia/reperfusion injury models?
Population
Rat model of lung ischemia/reperfusion injury and human pulmonary artery endothelial cells-based…
Design
Preclinical
Follow-up
2 hours of reperfusion (in vivo)
Authors
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May protect against experimental lung I/R injury; extends preclinical EET data but leaves open human translation.
Does CYP2J2 overexpression or exogenous EETs reduce oxidative stress and apoptosis in lung ischemia/reperfusion injury models?
CYP2J2 overexpression and exogenous EETs protect against oxidative stress and apoptosis in lung ischemia/reperfusion injury, suggesting a potential therapeutic strategy.
Chen et al. (2014) studied this question.
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