Key result
A 57-year-old man with COVID-19 pneumonia developed a transient type 1 Brugada ECG pattern without ongoing fever, which completely resolved at 1-month follow-up.
Why the study?
Patients with COVID-19 pneumonia can develop cardiac complications and may be susceptible to proarrhythmia triggered by fever, stress, electrolyte disturbances, or antiviral drugs.
Case Report (n=1)
No
SARS-CoV-2 infection may induce a transient Type 1 Brugada ECG pattern even in the absence of fever, suggesting a potential direct viral effect on myocardial ion channels.
May signal proarrhythmic susceptibility in afebrile COVID-19; single case leaves open clinical significance and need for validation.
Patients with COVID-19 pneumonia can develop heart problems and may also may be susceptible to proarrhythmia, virus-related issues such as fever, stress, electrolyte disturbance and adverse effects from the use of antiviral drugs. We report a transient Brugada-like ECG pattern without ongoing fever in a 57-year-old man, admitted with a diagnosis of COVID-19 pneumonia, who did not have syncope or a family history of sudden cardiac death. LEARNING POINTS: Patients with COVID-19 pneumonia can develop heart problems.A transient Brugada-like ECG pattern was observed in a non-febrile COVID-19 patient.SARS-CoV-2 may have a direct effect on myocardial ion channels.
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Pasqualetto et al. (2020) conducted a case report in COVID-19 pneumonia (n=1). COVID-19 infection was evaluated on ECG changes (Brugada pattern). A 57-year-old man with COVID-19 pneumonia developed a transient type 1 Brugada ECG pattern without ongoing fever, which completely resolved at 1-month follow-up.
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