Population
Mice with early cardiac hypertrophy induced by transverse aortic constriction and transgenic mice expressing…
Design
Preclinical
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Potential compensatory mechanism in early hypertrophy; hypothesis-generating for PDE-targeted cGMP modulation in human HF.
Redistribution of cGMP-regulated phosphodiesterases in early cardiac hypertrophy augments cGMP-mediated contractility, representing a potential compensatory mechanism to sustain heart function.
Perera et al. (2015) studied this question.
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