Key result
Swimming exercise induced a highly significant increase in 3H-thymidine incorporation in capillary wall cells, suggesting significant neoformation of myocardial blood vessels.
Physiological cardiac hypertrophy induced by swimming exercise is associated with significant neoformation of myocardial blood vessels, whereas pathological hypertrophy models show minor or no neoformation.
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Supports angiogenesis in exercise-induced hypertrophy in rats; leaves open relevance to human physiological vs pathological remodeling.
Ljungqvist et al. (1973) studied Experimental cardiac hypertrophy. Swimming exercise, arterial hypertension, and aortic stenosis vs. Rest period, short-term hypertension, or 2-month aortic stenosis was evaluated on Nuclear incorporation of 3H-thymidine (proliferative activity). Swimming exercise induced a highly significant increase in 3H-thymidine incorporation in capillary wall cells, suggesting significant neoformation of myocardial blood vessels.
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