Reserpine-induced catecholamine depletion reduces ventricular escape during vagal stimulation, highlighting the role of catecholamines in this electrophysiological response.
Hypothesis-generating for catecholamine facilitation of ventricular escape in animals; clinical relevance to human vagal arrhythmias remains undetermined.
Catecholamine depletion by reserpine diminishes the tendency of the ventricle to escape from vagal suppression. Neither spinal section nor adrenalectomy enhances the reserpine effect. Norepinephrine restores the characteristic occurrence of ventricle escape during vagal stimulation.
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Roberts et al. (1960) studied this question.
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