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Case report reveals significant hyperkalaemia following indomethacin therapy in mild chronic renal failure, indicating a drug-induced defect in cellular potassium uptake.
A patient with mild chronic renal failure developed significant hyperkalaemia while receiving indomethacin therapy. The hyperkalaemia, which was reversed by discontinuing indomethacin, is attributed to a drug-induced defect in the cellular uptake of potassium.
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MacCarthy et al. (1979) studied this question.
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