Key result
Primary PCI in STEMI patients led to an immediate increase in platelet microparticles and a significant decrease in leukocyte- and endothelial-derived microparticles, which subsequently increased over 48 hours.
Why the study?
What is the time course of changes in circulating microparticles of different cell origins in STEMI patients undergoing primary PCI?
Population
24 patients with ST-segment elevation myocardial infarction undergoing primary percutaneous transluminal…
Design
Cohort
Follow-up
48 hours
Authors
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Microparticle shifts after primary PCI in STEMI may reflect distinct cellular roles; leaves open biomarker utility pending prospective trials.
Observational (n=24)
No
What is the time course of changes in circulating microparticles of different cell origins in STEMI patients undergoing primary PCI?
Absolute Event Rate: 114% vs 250%
p-value: p=0.01
Circulating microparticles of different cellular origins exhibit distinct dynamic changes following primary PCI in STEMI patients, which may reflect their different pathophysiological roles.
Zhou et al. (2016) conducted an observational in ST-segment elevation myocardial infarction (STEMI) (n=24). Percutaneous transluminal coronary intervention (PCI) vs. Baseline (pre-PCI) was evaluated on Leukocyte-derived microparticle (LMP) levels immediately after PCI compared to baseline (p=0.01). Primary PCI in STEMI patients led to an immediate increase in platelet microparticles and a significant decrease in leukocyte- and endothelial-derived microparticles, which subsequently increased over 48 hours.
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