Key result
Lipopolysaccharide pretreatment before cecal ligation and perforation prevented peritonitis-induced cardiac myocytes from promoting polymorphonuclear leukocyte migration and activating endothelial cells.
Why the study?
Does lipopolysaccharide pretreatment prevent peritonitis-induced proinflammatory changes in cardiac myocytes in rats?
Does lipopolysaccharide pretreatment prevent peritonitis-induced proinflammatory changes in cardiac myocytes in rats?
Lipopolysaccharide pretreatment abrogates the systemic response that induces cardiac myocytes to become proinflammatory following peritonitis in a rat model.
Answers are grounded in this paper and cite their sources.
Hypothesis-generating in rat peritonitis model; does not support clinical use of lipopolysaccharide pretreatment.
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Madorin et al. (2001) studied Peritonitis-induced myocardial inflammation. Lipopolysaccharide pretreatment vs. No pretreatment was evaluated on Polymorphonuclear leukocyte transendothelial migration and intercellular adhesion molecule-1 surface expression. Lipopolysaccharide pretreatment before cecal ligation and perforation prevented peritonitis-induced cardiac myocytes from promoting polymorphonuclear leukocyte migration and activating endothelial cells.
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