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In vitro study demonstrates lipopolysaccharide impairs cellular respiration in rat cardiac endothelial cells, highlighting a pathway for sepsis-induced vascular damage.
Endothelial injury and dysfunction are key patho-physiological processes in sepsis. The aim of the study was to evaluatethe effects of bacterial lipopolysaccharide (LPS) on cellular respiration of rat primary cardiac microvascular endothelial cells (CMEC).
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Vuda et al. (2011) studied this question.
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