Key result
Arterial carbon dioxide tension (PaCO2) influences myocardial inotropy, a relationship that can be characterized non-invasively using the squared reciprocal of the pre-ejection period (1/PEP2).
Why the study?
How do changes in arterial carbon dioxide tension affect the squared reciprocal of the pre-ejection period (1/PEP2) as a non-invasive inotropic index?
How do changes in arterial carbon dioxide tension affect the squared reciprocal of the pre-ejection period (1/PEP2) as a non-invasive inotropic index?
The squared reciprocal of the pre-ejection period (1/PEP2) may serve as a non-invasive inotropic index to evaluate myocardial response to changes in PaCO2.
Answers are grounded in this paper and cite their sources.
1/PEP2 may non-invasively track PaCO2 effects on inotropy; hypothesis-generating and should not yet change practice.
The squared reciprocal of the pre-ejection period (1/PEP2) is an inotropic index which may be measured without catheterization of the left ventricle. Changes in 1/PEP2 in response to changes in arterial carbon dioxide tension have been studied. The character of the Paco2/myocardial inotropy response
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Blackburn et al. (1972) studied this question. Arterial carbon dioxide tension (PaCO2) influences myocardial inotropy, a relationship that can be characterized non-invasively using the squared reciprocal of the pre-ejection period (1/PEP2).
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