Key result
Static stretching after resistance training prevents acute post-exercise impairments in FMD and HRV.
Why the study?
Resistance training may induce transient changes in endothelial and autonomic patterns, but whether post-exercise static stretching modulates acute endothelial, autonomic, and haemodynamic recovery compared with resistance training alone remains unclear.
Does static stretching after resistance training improve acute endothelial, autonomic, and hemodynamic recovery in healthy young males?
RCT (n=27)
Open-label
Simple randomization via online tool
No
Does static stretching after resistance training improve acute endothelial, autonomic, and hemodynamic recovery in healthy young males?
Standardized Mean Difference: 0.9
p-value: p=0.012
The addition of static stretching after resistance training promotes faster recovery of endothelial function and autonomic modulation compared to resistance training alone in healthy young males.
Adding static stretching after resistance training prevents prolonged endothelial and autonomic impairment; extends acute recovery data beyond resistance training alone in healthy men.
Purpose Resistance training (RT) may induce transient changes in endothelial and autonomic patterns. Static stretching (SS) performed at the end of an RT session may alter the temporal course of recovery of these responses. This study investigates whether SS following RT modulates acute endothelial, autonomic, and haemodynamic recovery compared with RT alone. Methods Twenty-seven healthy males completed three experimental sessions in a randomized-crossover design: RT, RT + SS, and control condition (CC). RT consisted of two upper-body resistance exercises (bench-press and seated-biceps curl), while RT + SS included the same RT protocol followed by SS of the same muscle groups. Flow-mediated dilation (FMD%), brachial-artery diameter (BA diameter), heart rate variability (HRV), heart rate (HR), and mean arterial pressure (MAP) were measured at baseline (BL), immediately after exercise (POST), and 30 min after exercise (POST30). Results RT produced a significant reduction in FMD% at POST ( p < 0.001; Cohen’s d = 1.58), which remained below the BL value at POST30 ( p = 0.012; d = 0.90); these decreases were not observed in RT + SS. The BA diameter increased following both conditions; however, at POST30 it remained above BL only following RT ( p < 0.001; d = 0.60). At POST, both conditions significantly reduced RMSSD and HF, along with an increase in HR. At POST30, however, HF and RMSSD showed an unfavorable recovery in RT alone ( p = 0.016; d = 0.72; p = 0.028; d = 0.57), while in RT + SS all values returned to BL. MAP decreased at POST in both conditions and returned to BL at POST30. Conclusion The addition of SS at the end of a RT session was associated with earlier autonomic and vascular recovery compared with RT alone. SS may represent a simple and sustainable recovery strategy after RT.
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Frenda et al. (2026) conducted an RCT in Healthy (n=27). Resistance training with post-exercise static stretching vs. Resistance training alone and seated control was evaluated on Flow-mediated dilation (FMD%) at 30 minutes post-exercise compared to baseline (Cohen's d = 0.90, p=0.012). The addition of static stretching after resistance training prevented the prolonged reductions in flow-mediated dilation and heart rate variability observed 30 minutes after resistance training alone.
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