Key result
A 57-year-old female with cardiac hypereosinophilia developed recurrent mechanical mitral valve thrombosis despite being on coumadin with a supratherapeutic INR, highlighting the potential inadequacy of standard anticoagulation in this population.
Why the study?
Hypereosinophilia can cause multiorgan damage and cardiac thrombus, but questions remain regarding optimal anticoagulation and management during atypical presentations.
Design
Case report
Authors
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May signal anticoagulation failure risk in cardiac hypereosinophilia; leaves open optimal regimens for mechanical valves.
Case Report (n=1)
No
Cardiac hypereosinophilia can lead to recurrent mechanical valve thrombosis even in the setting of supratherapeutic INR on coumadin, suggesting alternative anticoagulation strategies may be needed.
Ostojić et al. (2022) conducted a case report in Cardiac hypereosinophilia with recurrent mechanical valve thrombosis (n=1). Coumadin was evaluated. A 57-year-old female with cardiac hypereosinophilia developed recurrent mechanical mitral valve thrombosis despite being on coumadin with a supratherapeutic INR, highlighting the potential inadequacy of standard anticoagulation in this population.
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