Analysis of the role of serum IL-2R, IL-6, IL-8, IL-10, and TNF-α in the treatment of immune escape mechanisms in patients with minimal ma tumors

Tumor immune escape refers to the phenomenon that tumor cells evade the recognition and attack of the body's immune system through a series of mechanisms, thus surviving and multiplying in the body. These mechanisms include, but are not limited to, low immunity, recognition as autologous antigen, antigenic modulation, tumor-induced immunosuppressive effects, and areas of tumor-induced generation immunity. Tumor cells often have low immunogenicity, i. e. they lack sufficient epitopes to stimulate the body's immune system. In addition, tumor cells can also produce immune suppressive molecules or cells by interacting with host cells, thus inhibiting the body's immune response. In addition, tumor cells can also produce immune suppressive molecules or cells by interacting with host cells, thus inhibiting the body's immune response. In this paper, the role of serum IL-2R, IL-6, IL-8, IL-10 and 1 TNF- α in the treatment of immune escape mechanism in patients was analyzed for reference.