Key result
Mutations in the N-terminus of the VSV M protein differentially affected association with plasma membranes and nucleocapsids, while mutations in the 75-106 region mainly affected nucleocapsid incorporation.
The study identifies distinct functional regions of the VSV M protein required for membrane association, nucleocapsid binding, and an additional unknown activity essential for viral replication.
Viral matrix (M) proteins bind the nucleoprotein core (nucleocapsid) to host membranes during the process of virus assembly by budding. Previous studies using truncated M proteins had implicated the N-terminal 50 amino acids of the vesicular stomatitis virus M protein in binding both membranes and nucleocapsids and a sequence from amino acids 75-106 as an additional membrane binding region. Structure-based mutations were introduced into these two regions, and their effects on membrane association and incorporation into nucleocapsid-M protein complexes were determined using quantitative assays. The results confirmed that the N terminus of M protein is involved in association with plasma membranes as well as nucleocapsids, although these two activities were differentially affected by individual mutations. Mutations in the 75-106 region affected incorporation into nucleocapsid-M complexes but had only minor effects on association with membranes. The ability of site-specific mutant M proteins to complement growth of temperature-sensitive M mutant virus did not correlate well with the ability to associate with membranes or nucleocapsids, suggesting that complementation involves an additional activity of M protein. Mutants with similar abilities to associate with membranes and nucleocapsids but differing in complementation activity were incorporated into infectious cDNA clones. Infectious virus was repeatedly recovered containing mutant M proteins capable of complementation but was never recovered with mutant M proteins that lacked complementation activity, providing further evidence for a separate activity of M protein that is essential for virus replication.
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Dancho et al. (2008) studied Vesicular stomatitis virus. Structure-based mutations in the N-terminal and 75-106 regions of VSV M protein was evaluated on Membrane association, incorporation into nucleocapsid-M protein complexes, and complementation of virus growth. Mutations in the N-terminus of the VSV M protein differentially affected association with plasma membranes and nucleocapsids, while mutations in the 75-106 region mainly affected nucleocapsid incorporation.
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