Key result
Adenovirus-linked cytotoxic corpus callosum lesions present as self-limiting pediatric encephalopathy with acute status epilepticus.
Adenoviral infection can be associated with cytotoxic lesions of the corpus callosum, presenting as a self-limiting cause of pediatric encephalopathy.
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Acute febrile encephalopathy progressing to status epilepticus in a toddler requires prompt intensive seizure control; leaves open etiology and optimal regimen.
A 30-month-old girl was admitted with an acute febrile illness, followed after 2 days by acute encephalopathy and recurrent seizures that rapidly escalated to status epilepticus, requiring continuous intravenous midazolam and levetiracetam administration.
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Ciaccio et al. (2026) studied this question. Cytotoxic lesions of the corpus callosum associated with adenoviral infection represent a self-limiting cause of pediatric encephalopathy that can present with acute status epilepticus.
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