Key result
NSAIDs or aspirin are linked to ~66% of bleeding peptic ulcers in acute GI bleeds.
Population
110 patients admitted for hematemesis or melena during a 6-month period in northern Greece.
Design
Cohort
Authors
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Supports NSAID/aspirin and H. pylori as key etiologies for peptic ulcer bleeding; leaves open whether targeted interventions improve outcomes.
Observational (n=110)
NSAID/low-dose aspirin use and H. pylori infection are major etiologic factors for acute non-malignant upper gastrointestinal bleeding in northern Greece.
Tsesmeli et al. (2006) conducted an observational in acute non-malignant upper gastrointestinal bleeding (ANMUGIB) (n=110). NSAIDs, low-dose aspirin, and Helicobacter pylori infection was evaluated on Etiology and sources of acute non-malignant upper gastrointestinal bleeding. Among 110 patients with acute non-malignant upper GI bleeding, bleeding peptic ulcers were the most frequent source, with 65.57% related to NSAIDs or low-dose aspirin and 60.65% to H. pylori.
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