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Demonstrates reduced myocardial uptake of tritiated digoxin in acutely hyperkalaemic dogs, indicating potassium-dependent inhibition of glycoside binding.
The distribution of tritiated digoxin in canine tissues in normal and acutely hyperkalaemic states is described. The reasons for the reduced myocardial uptake of the glycoside in the presence of hyperkalaemia are discussed.
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Morgan et al. (1970) studied this question.
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