Key result
Disulfiram inhibited cardiomyocyte viability and promoted apoptosis, with cardiomyocytes demonstrating greater sensitivity to the drug than cancer cells.
Why the study?
Does disulfiram inhibit viability and induce apoptosis in cardiomyocytes?
Population
cardiomyocytes and cancer cells
Design
Preclinical
Authors
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May signal disulfiram cardiotoxicity risk; animal data leave open human relevance and mitigation.
Does disulfiram inhibit viability and induce apoptosis in cardiomyocytes?
Disulfiram exhibits potential cardiotoxic effects by inhibiting viability and inducing apoptosis in cardiomyocytes, though PEG may serve as a promising solvent to mitigate these adverse effects.
Li et al. (2018) studied Cardiomyocyte viability and apoptosis. Disulfiram (DSF) vs. Cancer cells was evaluated on Cardiomyocyte viability and apoptosis. Disulfiram inhibited cardiomyocyte viability and promoted apoptosis, with cardiomyocytes demonstrating greater sensitivity to the drug than cancer cells.
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