Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
September 1, 2007Cancer ResearchOpen Access

Requirement for Rac1 in a K-ras–Induced Lung Cancer in the Mouse

View Full Paper
Ask AI
Bookmark
Share

Authors

JKJoseph L. KissilThe Wistar InstituteMWMarita J. WalmsleyCentre National de la Recherche ScientifiqueLHLinda HanlonThe Wistar Institute

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Kissil et al. (2007) studied this question.

synapsesocial.com/papers/6aa78c4bd0fe1e5ca4d13184https://doi.org/10.1158/0008-5472.can-07-2300
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Rho GTPases are over-expressed in human tumors1999 · 670 citations
  2. 2p21-Activated Kinase 1 Phosphorylates the Death Agonist Bad and Protects Cells from Apoptosis2000 · 358 citations
  3. 3Hyperactivation of P21ras and the Hematopoietic-Specific Rho Gtpase, Rac2, Cooperate to Alter the Proliferation of Neurofibromin-Deficient Mast Cells in Vivo and in Vitro2001 · 135 citations
  4. 4Oncogenic Ras Downregulates Rac Activity, Which Leads to Increased Rho Activity and Epithelial–Mesenchymal Transition2000 · 289 citations