Key result
Chest wall stimulation reliably terminates magnet-unresponsive endless loop tachycardia by inhibiting the DDD ventricular channel.
Why the study?
Endless loop tachycardia in DDD pacing is typically terminated by magnet application, but some cases are magnet unresponsive due to AV desynchronization arrhythmia.
Does chest wall stimulation terminate magnet unresponsive endless loop tachycardia in patients with DDD pacemakers?
Does chest wall stimulation terminate magnet unresponsive endless loop tachycardia in patients with DDD pacemakers?
Chest wall stimulation provides a reliable method to terminate endless loop tachycardia in DDD pacemakers when magnet application fails and programmers are unavailable.
May offer bedside termination when magnets fail and programmers unavailable; leaves open validation in modern DDD devices.
Endless loop tachycardia is a well-known complication of DDD pacing and is almost invariably terminated by conversion to the asynchronous DOO mode upon application of a magnet over the pulse generator. Occasionally magnet application is ineffectual because the ventriculoatrial (VA) synchrony of endless loop tachycardia is converted directly or indirectly to an atrioventricular (AV) desynchronization arrhythmia, another form of VA synchrony. This occurs when a paced ventricular beat engenders an unsensed retrograde P wave and the continual delivery of an ineffectual atrial stimulus during the atrial myocardial refractory period creates self-perpetuating VA synchrony. Upon magnet removal, AV desynchronization arrhythmia reverts immediately to endless loop tachycardia. In the absence of access to programmers, magnet unresponsive endless loop tachycardia can be easily and reliably terminated by chest wall stimulation through inhibition of the ventricular channel of the DDD pulse generator.
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Barold et al. (1988) studied Endless loop tachycardia. Chest wall stimulation was evaluated on Termination of endless loop tachycardia. Chest wall stimulation easily and reliably terminates magnet unresponsive endless loop tachycardia by inhibiting the ventricular channel of the DDD pulse generator.
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