Key result
PCOS is linked to ~96% higher CRP levels versus healthy controls, highlighting chronic low-grade inflammation.
Why the study?
The role of inflammatory mediators and their genetic mechanisms in the pathogenesis of chronic low-grade inflammation in PCOS is not fully elucidated.
Population
Reproductive-age women with PCOS
Design
Review
Authors
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May inform PCOS inflammation assessment; leaves open whether targeting CRP alters outcomes.
Chronic low-grade inflammation and genetic polymorphisms in inflammatory mediators play a significant role in the pathogenesis and metabolic manifestations of PCOS.
Deligeoroglou et al. (2012) conducted a review in Polycystic ovarian syndrome (PCOS). Inflammatory mediators vs. Healthy controls was evaluated. C-reactive protein levels are 96% higher in PCOS patients than in healthy controls, highlighting the role of chronic low-grade inflammation in the syndrome's pathogenesis.
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