Key result
Charged residues in Csm3 dictate crRNA maturation cleavage while Csm5 regulates non-Cas cellular nucleases.
Why the study?
The molecular mechanisms underlying crRNA maturation in the Cas10–Csm complex and the roles of non-Cas nucleases remain unclear.
The study elucidates the roles of Csm3, Csm5, and non-Cas nucleases in crRNA maturation within the Type III-A CRISPR-Cas system.
Csm3's role in Type III-A crRNA maturation leaves open applications in CRISPR engineering; prospective studies needed before any clinical translation.
CRISPR–Cas (Clustered regularly interspaced short palindromic repeats-CRISPR-associated proteins) is a prokaryotic immune system that destroys foreign nucleic acids in a sequence-specific manner using Cas nucleases guided by short RNAs (crRNAs). Staphylococcus epidermidis harbours a Type III-A CRISPR–Cas system that encodes the Cas10–Csm interference complex and crRNAs that are subjected to multiple processing steps. The final step, called maturation, involves a concerted effort between Csm3, a ruler protein in Cas10–Csm that measures six-nucleotide increments, and the activity of a nuclease(s) that remains unknown. Here, we elucidate the contributions of the Cas10–Csm complex toward maturation and explore roles of non-Cas nucleases in this process. Using genetic and biochemical approaches, we show that charged residues in Csm3 facilitate its self-assembly and dictate the extent of maturation cleavage. Additionally, acidic residues in Csm5 are required for efficient maturation, but recombinant Csm5 fails to cleave crRNAs in vitro. However, we detected cellular nucleases that co-purify with Cas10–Csm, and show that Csm5 regulates their activities through distinct mechanisms. Altogether, our results support roles for non-Cas nuclease(s) during crRNA maturation and establish a link between Type III-A CRISPR–Cas immunity and central nucleic acid metabolism.
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Walker et al. (2016) studied CRISPR-Cas immunity in Staphylococcus epidermidis. Genetic and biochemical modifications of Csm3 and Csm5 was evaluated on crRNA maturation and cleavage. Genetic and biochemical analyses revealed that charged residues in Csm3 dictate maturation cleavage, while Csm5 regulates non-Cas cellular nucleases during crRNA maturation.
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