Key result
iNOS-derived NO may act as a signaling molecule targeting cysteine-Zn2+ linkages during nitrosative stress.
Why the study?
The effects of inducible nitric oxide synthase-derived NO on proteins containing cysteine-Zn2+ complexes and their cellular roles during nitrosative stress are not fully understood.
Design
Review
Authors
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NO nitrosation mechanisms in inflammation extend basic understanding; leaves open any clinical translation or therapeutic targeting.
Key points are not available for this paper at this time.
Klaus-D. Kröncke (2001) reported a review. Inducible nitric oxide synthase-derived NO is suggested to act as a signal molecule targeting cysteine-Zn2+ linkages, enabling cells to react toward nitrosative stress.
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