Key result
High-output heart failure is an uncommon condition driven by reduced systemic vascular resistance, which can be aggravated by an arteriovenous fistula for hemodialysis access.
This editorial discusses the pathophysiology of high-output heart failure, specifically focusing on reduced systemic vascular resistance caused by conditions such as arteriovenous fistulas for hemodialysis.
High-output heart failure is an uncommon type of acute heart failure, and is caused by various pathological conditions, such as anemia, sepsis, beriberi, congenital or acquired arteriovenous fistula, hyperthyroidism, psoriasis, and Paget disease of bone [1,2]. The underlying pathophysiological condition in high-output heart failure is reduced systemic vascular resistance, which is elicited by either vasodilation of systemic resistant vessels or systemic arteriovenous shunt. The decreased systemic vascular resistance activates hemodynamic and neurohormonal responses that maintain blood supply to the organs.
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Yutaka Kagaya (2015) conducted an editorial in High-output heart failure. Arteriovenous fistula for hemodialysis access was evaluated. High-output heart failure is an uncommon condition driven by reduced systemic vascular resistance, which can be aggravated by an arteriovenous fistula for hemodialysis access.
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