Key result
Real-time invasive hemodynamics identifies high-output heart failure from arteriovenous access.
Case Report
Consider high-output HF in ESRD patients with AV access and unexplained CHF; hypothesis-generating for access modification, pending prospective data.
Congestive heart failure (CHF) is an important source of morbidity and mortality in end-stage renal disease patients. Although CHF is commonly associated with low cardiac output (CO), it may also occur in high CO states. Multiple conditions are associated with increased CO including congenital or acquired arteriovenous fistulae or arteriovenous grafts. Increased CO resulting from permanent AV access in dialysis patients has been shown to induce structural and functional cardiac changes, including the development of eccentric left ventricle hypertrophy. Often, the diagnosis of high output heart failure requires invasive right heart monitoring in the acute care setting such as a medical or cardiac intensive care unit. The diagnosis of an arteriovenous access causing high output heart failure is usually confirmed after the access is ligated surgically. We present for the first time, a case for real-time hemodynamic assessment of high output heart failure due to AV access by interventional nephrology in the cardiac catheterization suite.
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Singh et al. (2014) conducted a case report in High output heart failure due to arteriovenous access in dialysis patients. Real-time hemodynamic assessment was evaluated. Real-time hemodynamic assessment of high output heart failure due to arteriovenous access was presented as a novel diagnostic approach in the cardiac catheterization suite.