Key result
Persistent myocardial inflammation drives pathogenesis of post-COVID cardiomyopathy and HFpEF.
Why the study?
Persistent myocardial inflammation is believed to play a central role in adverse left ventricular remodeling and the pathogenesis of dilated cardiomyopathy and HFpEF, but its role in post-COVID cardiomyopathy requires further understanding.
Discusses the role of myocardial inflammation in the development of heart failure, particularly HFpEF, in the context of COVID-19.
Answers are grounded in this paper and cite their sources.
Myocardial inflammation may link COVID-19 to HFpEF; hypothesis-generating and should not yet change practice.
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Carvalho et al. (2023) conducted a review in Post-COVID Cardiomyopathy and Heart Failure. COVID-19 / Myocardial inflammation was evaluated. Persistent myocardial inflammation is believed to play a central role in the pathogenesis of post-COVID cardiomyopathy and heart failure with preserved ejection fraction.
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