Key result
Middle sacral artery ligation successfully resolves refractory type II endoleak after prior embolization failures.
Why the study?
Type II endoleak after endovascular aortic aneurysm repair remains difficult to identify and treat, especially when associated with continuous aneurysm sac expansion.
Case Report (n=1)
Type II endoleaks after EVAR can be fed by difficult-to-identify sources such as the middle sacral artery, highlighting the need for careful diagnostic planning and management.
Persistent type II endoleak with sac expansion remains diagnostically challenging; leaves open optimal imaging strategy after EVAR.
Type II endoleak after endovascular aortic aneurysm repair still remains the Achilles' heel of the treatment, the source of which regularly is difficult to identify and treat. We present a patient with a persistent type II endoleak associated with a continuous aneurysm sac expansion after endovascular aortic aneurysm repair for which many diagnostic modalities were used during his follow-up such as duplex scan, computed tomography angiography and magnetic resonance angiography. Attempts were undertaken to treat the source of endoleak including coil micro-embolisation of lumbar arteries and subsequent open ligation of the inferior mesenteric artery, but they failed to eliminate the endoleak. Finally, a middle sacral artery was identified as the source of the endoleak. At that time, the patient was subjected to surgery for sigmoid carcinoma, and simultaneously, a ligation of the sacral artery was undertaken which eventually eliminated the endoleak completely. This case highlights that type II endoleak may be evoked by various sources and there can be a great difficulty to identify these feeding vessels; thus, careful planning for its management is mandatory.
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Spanos et al. (2014) conducted a case report in Persistent type II endoleak after endovascular abdominal aortic aneurysm repair (n=1). Ligation of the middle sacral artery was evaluated on Elimination of endoleak. Ligation of the middle sacral artery successfully eliminated a persistent type II endoleak that had failed previous coil micro-embolisation and inferior mesenteric artery ligation.
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