Key result
COVID-19 vaccination carries a substantially lower risk of myocarditis than natural SARS-CoV-2 infection.
Why the study?
Chronic heart failure is a major global burden with respiratory viral infections frequently precipitating hospitalization and mortality, and viral myocarditis significantly contributes to cardiac dysfunction through direct and immune-mediated mechanisms.
This comprehensive review emphasizes that the rare risk of mild vaccine-associated myocarditis is heavily outweighed by the significantly higher risk of severe cardiac complications from natural SARS-CoV-2 infection.
Chronic heart failure remains a major global burden, with respiratory viral infections frequently precipitating hospitalisation and mortality.Viral myocarditis has emerged as a significant contributor to cardiac dysfunction, driven by both direct cytopathic effects and immune-mediated pathways.This review synthesises contemporary experimental and clinical evidence on the molecular mechanisms by which cardiotropic viruses-including Coxsackievirus B, herpesviruses (HHV 6, EBV, CMV), parvovirus B19, influenza viruses and SARS CoV 2damage the myocardium, with particular emphasis on the roles of Toll like receptors, viral proteases and the pro inflammatory cytokines TNF α, IL 6 and IL 1β.These cytokines exert negative inotropic actions, promote cardiomyocyte apoptosis and disrupt extracellular matrix homeostasis, thereby contributing to adverse ventricular remodelling and heart failure progression.The review also examines the veterinary legacy of coronavirus vaccine development, which has been largely overlooked in human cardiology.For over half a century, veterinary medicine has confronted coronaviruses as devastating livestock pathogens, accumulating extensive knowledge of vaccine platforms, viral mutation and immune escape mechanisms that proved instrumental in expediting the creation of mRNA vaccines against COVID 19.Importantly, the review appraises the cardiac safety profile of these vaccines, concluding that the incidence of vaccine associated myocarditis-predominantly observed in young males after the second dose-is substantially lower than the risk of myocarditis following natural SARS CoV 2 infection.Although rare, post vaccination cardiac complications warrant ongoing surveillance and mechanistic research.Overall, this comprehensive overview underscores the convergence of infectious disease, immunology and vaccinology in shaping our understanding of virus related heart disease, while highlighting the translational value of veterinary research.Future investigations should focus on identifying individual susceptibility factors and developing targeted strategies to mitigate both viral and vaccine induced myocardial injury, thereby improving outcomes for patients with or at risk of heart failure.
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Tokmachev et al. (2026) conducted a review in Viral myocarditis and post-vaccination myocardial injury. Viral infection and mRNA COVID-19 vaccines was evaluated. Viral myocarditis is driven by direct cytopathic effects and immune-mediated pathways, and the risk of myocarditis following COVID-19 vaccination is substantially lower than following natural infection.
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