August 4, 2025

Table S4 from VAV2 Drives EGFR-Mediated Rac1 Responses in Prostate Cancer

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MAIN FINDING: VAV2 significantly drives EGFR-mediated Rac1 responses in prostate cancer cells.
KEY EVIDENCE: Analysis of Rac-GEF expression in human prostate adenocarcinoma shows notable levels of Rac1 activity.
APPROACH: The study examines the role of VAV2 in regulating signaling pathways in prostate cancer models.
SIGNIFICANCE: Targeting VAV2 and its pathways may offer innovative strategies for treating prostate adenocarcinoma.

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Findings reveal VAV2 influences EGFR-mediated Rac1 activation in prostate cancer, suggesting new therapeutic targets.

Table S4. Expression of Rac-GEFs in human prostate adenocarcinoma.

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Baker et al. (2025) studied this question.

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Findings reveal VAV2 influences EGFR-mediated Rac1 activation in prostate cancer, suggesting new therapeutic targets.

Table S4. Expression of Rac-GEFs in human prostate adenocarcinoma.